A molecular mechanism involving the tight balance between two proteins — Gal-1 and CaV1.2 — was found to regulate the behavior of blood vessel cells, directly contributing to hypertension.
This finding provides new insights into the underlying processes that trigger arterial contraction and hypertension, and its researchers suggest that a treatment based on Gal-1 might offer a “cost-effective” means of controlling blood pressure and pulmonary arterial hypertension.
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